Japanese scientists show administering NMN to aged men more than doubles blood NAD+ levels and enhances walking speed and grip strength.
Chinese scientists find that NMN restores the age-related deterioration of the intestinal wall by boosting natural antioxidant defenses and reducing inflammation in older mice.
Scientists show that increasing the abundance of an NMN transporter in the brain of mice counters the age-related decline in muscle strength, endurance, and metabolism.
Researchers in China show that the NAD+ precursor NMN and antioxidant resveratrol boosts the cognition of aged rats by their senolytic action.
Italian scientists find that the anti-aging compound resveratrol mitigates markers of aging, including abnormal heart rhythms and senescent cells in a human heart model.
By improving mitochondrial production and functional capacity, NMN with troxerutin fully protects the heart against the toxic effects of the chemotherapeutic doxorubicin in rats.
Administering NMN with resveratrol synergistically boosts NAD+ in the heart and skeletal muscle of mice, while combining NMN with ginsenosides amplifies NAD+ in the lungs.
Treating obese female mice with nicotinamide mononucleotide (NMN) reduces DNA damage and improves structural protein composition within egg cells (oocytes) while restoring low offspring birth weight.
Chinese scientists find that boosting NAD+ with its precursor, NMN improves the learning and memory of a mouse model for Alzheimer’s disease.
Harvard researchers introduce the concept of banking fecal matter during our healthy years to rejuvenate our gut microbes later in life and treat age-related disease.
Mitobridge researchers find that heart tissue death can be minimized by boosting NAD+ with a drug called MK-0159, designed to block an immune cell protein called CD38.
A new study finds that NMN improves the metabolism, hormone levels, and skin of postmenopausal women.
A drug that lowers blood sugar (empagliflozin) improves age-related blood vessel dysfunction associated with cardiovascular disease in mice.
Recent failed clinical trials for the treatment of Alzheimer’s point to stifled innovation, beckoning for prevention strategies for the generations to come.
The anti-aging compounds NMN, metformin, and rapamycin prevent brain damage and alleviate memory deficiencies in a rat model for age-related cognitive impairment.